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Cyclin B-Cdc2 complex / maturation promoting factor: activation by both phosphorylation (cytoplasmic->nuclear localization) and dephosphorylation (at different sites)
- downstream events: phosphorylation of kinesin-like motor protein (bipolar spindle formation) and nuclear lamins (disassembly of the nuclear envelope)
proton oncogenes: normal cellular genes that promoted cell proliferation or inhibit apoptosis. GOF mutations in proton-oncogenes are associated with cancer.
tumor-suppressor genes: normal function is to prevent cell division and promote apoptosis. LOF mutations in tumor-suppressor genes cause cancer
calculating allele frequencies: CCR5
AA: 0.795, Aa: 0.19, aa: 0.015
Allele freq: A = 1780/2000 = 0.89
a = 220/2000 = 0.11
A loss-of-function mutation in one of the listed genes is known to contribute to colorectal carcinomas. Which one?
A loss-of-function mutation in one of the listed genes is known to contribute to colorectal carcinomas. Which one?
Bcl2
Ras
Myc
Mdm2
Bax
Question at position 2
2
0 points
Question at position 2
The progression from G2 to M is controlled by
The progression from G2 to M is controlled by
cyclin D - Cdk4 complex
Bax-Bcl2 heterodimer complex
p53 - Mdm2 complex
Cyclin B - Cdc2 complex
Cyclin E - Cdk2 complex
Question at position 3
3
0 points
Question at position 3
A population has the following genotype frequencies:
AA: 0.36; Aa: 0.2; aa: 0.44. What is the frequency of the a allele?
A population has the following genotype frequencies:
AA: 0.36; Aa: 0.2; aa: 0.44. What is the frequency of the a allele?
0.80
0.64
0.56
0.194
0.54
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