Notes

EMF Blocking in addition to EMF Shielding
If you reside in an apartment or a house or simply need to ensure that your house is free of EMFs There are a variety of methods to reduce exposure. One of the most effective is to limit your electronic device use. You could also consider EMF block paint to prevent EMF radiation from entering your home. Another method to protect your home against EMF radiations is to put up a shielding canopy for RF. This is a fabric made of net that has EMF shielding and is used to block EMFs from entering rooms. Another alternative is to have your home fitted with a conductive enclosure. Proteck'd are known as Faraday cages.

Several studies have shown studies have shown that nonionizing EMF can cause antiproliferative properties in HCC cells. The mechanism of AM RF EMF's anticancer activity in vitro is believed to involve down-regulation the cancer stem cell. This could explain the long-term responses seen in some patients with advanced HCC. However, the mechanism of AM EMF's impact on cancer patients is not yet clear.


The effects on the effects of AM RF EMF on HCC tumor growth in vivo was studied in mice. The tumors were split into 3 groups. First, the group that was unaffected RF EMF. Another group of participants was subjected RF EMF at frequencies similar to the one used by humans. In the third, they were subjected RF EMF at HCC-specific modulation frequencies. The effects of HCCMF on tumours was compared to that of RCF. The results indicated that cancers treated with HCCMF were significantly shrinking. However, the tumors treated with RCF showed no evidence of shrinkage of the tumor.

The reason for tumour-specific AM RF EMF may be based on the fact that tumor cells require Cav3*2 voltage calcium channels for their proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation upon HCC cells is mediated by CACNA1H the protein that is responsible for the influx of Ca2+ specific to tumours. The results indicate that CACNA1H could have wider implications in the diagnosis and treatment of various cancers.

The tumors in those in the group that were unaffected to RF EMF, and were fed a normal mouse diet. The tumors of those in the HCCMF group were infected with Huh7 cells when they were five-seven weeks old. The tumors were then killed when they showed excessive burden.

The tumours in the three groups also displayed different growth curves. The HCCMF-treated tumors showed a significant decrease in the size of the tumour after eight weeks. However, the tumors treated with RCF didn't show shrinkage. The difference was substantial. The tumours treated with RCF had necrosis that is common when tumors are that are exposed to RCF. There is a possibility that this necrosis was due to an absence of oxygen in the more invasive tumours.

In sum, the results suggest an AM-RF EMF has anticancer properties in vitro as well as in vivo. Numerous studies have demonstrated the fact that AM RF EMF produces measurable shrinkage of tumors within HCC patients. It is possible that AM RF EMF triggers these effects through CACNA1H, a protein involved in tissue-specific Ca2+ influx. Additionally, AM RF EMF may exert a sustained impact on the growth of HCC tumors in vivo.

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