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No matter if you live in an apartment or house or simply would like to keep your home free of electromagnetic fields, there are a number of methods to reduce exposure. One of the easiest is to restrict the use of electronic devices. You can also turn to EMF block paint to prevent EMF radiation from reaching your home. Another easy way to protect your house against EMF radiation would be to put up a RF shielding canopy. This is a cloth of net that has EMF shielding. It's used to prevent EMFs from entering a space. Another option is to get your home fitted with an enclosure that is conductive. These enclosures are known as Faraday cages.
Several studies have shown how the EMF that is not ionized produces antiproliferative properties in HCC cells. The mechanism behind AM RF EMF's anticancer activity in vitro is believed to involve down-regulation in cancer-related stem cells. This may account for the long-term effects observed in some patients with advanced HCC. However, the mechanism of AM RF EMF's effect in patients with cancer is not clear.
Aspects on the effects of AM RF EMF on HCC tumor growth in vivo were studied in mice. The tumors were divided in three different groups. emf blocker did not have exposure RF EMF. Second group members were exposed RF EMF at frequencies similar to that of humans. In the third, they were subjected to the RF EMF in HCC-specific frequencies. The impact of HCCMF on tumours was assessed against the effect of RCF. The results indicated that cancers treated with HCCMF showed significant shrinkage. However, tumours treated with RCF showed no evidence of tumour shrinkage.
The mechanism of tumor-specific AM RF EMF might be driven by the fact that tumor cells require Cav3*2 type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's antiproliferative effects upon HCC cells is mediated through CACNA1H which is a protein which is responsible for the influx of Ca2+ specific to tumours. The results indicate that CACNA1H could have wider implications for the diagnosis and treatment of various cancers.
The tumors in those in the group that were unaffected to EMF from radiofrequency, and fed a normal mouse diet. The tumors in HCCMF HCCMF group were injected with Huh7 cells at the time they were 5 to 7 weeks old. The tumors were removed in cases of excessive burden.
The tumors of the three groups showed different growth curves. The tumours treated with HCCMF had a significant reduction in tumour size after 8 weeks. However, the tumours which were treated by RCF didn't show reduction in size. The difference was significant. The tumors treated with RCF had necrosis, which is common in tumours that are exposed to RCF. There is a possibility that the necrosis was caused by the lack of oxygen in the larger tumours.
In conclusion, the findings show the fact that AM EMF exhibits anti-cancer properties in vitro as well as in live. Several studies have shown it is true that AM RF EMF produces measurable tumour shrinkage within HCC patients. There is a possibility that AM RF EMF triggers these effects because of CACNA1H, a protein that is involved in the process of tissue-specific Ca2+ influx. Additionally, AM RF EMF may have a long-lasting influence on the growth of HCC tumours in the vivo.
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