Notes

The Nobel Prize event ignited this "Immortality" technology. The sky high price has passed, and everyone is almost 100 years old.
Phosphatidylserine,Anti-Aging

Following the world-famous Nobel Prize event, this year's winner of the chemistry prize, 97 year old John. B. the news heat of breaking the record of the "oldest" Nobel laureate is soaring. The Buddhist legends of "I retired at 102" and "50 is just the beginning" are even more difficult.
Good stock, 97, still goes to the laboratory to do scientific research every day.
While many people are still feeling that good stock is old and strong, the outstanding contributions of the three winners of this year's Nobel Prize in medicine have once again detonated the "Immortality" technology that people are most concerned about. In 2019, one of the biggest research hotspots in the field of life science is nicotinamide mononucleotide (i.e. now hot rich volume).
Three winners of this year's Nobel Prize in medicine
Hypoxia inducible factor-1 (HIF-1), the main scientific contribution of the three medical prize winners, is not only the "switch" to activate bone marrow to quickly adapt to the hypoxic environment, but also the "culprit" to cause pathological reactions such as aneurysms and cardiomyopathy and accelerate body aging under adverse conditions such as continuous accumulation.
Previous studies have shown that the pseudo hypoxic environment caused by the decrease of NAD content in aging cells is the key factor for the accumulation of HIF-1, but the level of human nad synthesis has been declining since the age of 30. Scientists began to study how to prolong life by breaking the limitation of this aging mechanism.
Nad content decreased, HIF-1 accumulated, and mitochondrial function was impaired (one of the signs of aging)
In 2013, consistent with the core mechanisms related to practical achievements such as levito, which is currently listed, this mining process made the first breakthrough in the laboratory of Harvard Medical School. David Sinclair, director of the Glenn biomedical Center for aging at Harvard University, published the research results in cell. The accumulation of HIF-1 was dramatically reversed by nad rebound of 22 month old mice supplemented with nicotinamide mononucleotide, and the mitochondrial function and skeletal muscle health level of elderly mice returned to the state of 6-month-old mice. More interestingly, in subsequent experiments, it was confirmed that this substance can prolong the life span of mammals similar to humans by more than 30%.
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